Global analysis of alternative splicing regulation by insulin and wingless signaling in Drosophila cells
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* Corresponding authors: Britta Hartmann britta.hartmann@crg.es - Juan Valcárcel juan.valcarcel@crg.es
1 Centre de Regulació Genòmica, Parc de Recerca Biomèdica de Barcelona, Dr. Aiguader 88, Barcelona, 08003, Spain
2 Universitat Pompeu Fabra, Parc de Recerca Biomèdica de Barcelona, Dr. Aiguader 88, Barcelona, 08003, Spain
3 Institut Municipal D'Investigació Mèdica, Parc de Recerca Biomèdica de Barcelona, Dr. Aiguader 88, Barcelona, 08003, Spain
4 Department of Molecular and Cell Biology, University of California, Berkeley, 94720, USA
5 Institució Catalana de Recerca i Estudis Avançats, Parc de Recerca Biomèdica de Barcelona, Dr. Aiguader 88, Barcelona, 08003, Spain
6 Current address: Stowers Institute for Medical Research, E. 50th Street, Kansas City, 64110, USA
7 Current address: Centre de Medicina Regenerativa de Barcelona, Parc de Recerca Biomèdica de Barcelona, Dr. Aiguader 88, Barcelona, 08003, Spain
Genome Biology 2009, 10:R11 doi:10.1186/gb-2009-10-1-r11
Published: 29 January 2009Additional files
Additional data file 1:
Calculated Z-scores values from the microarray experiments, for each biological replicate, for genes that are transcriptionally regulated by the insulin (top) and wingless (bottom) signaling pathways.
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Additional data file 2:
Calculated Z-scores for splice junction probes that are regulated upon induction of the insulin (top) and wingless (bottom) signaling pathways.
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Additional data file 3:
The table represents the percentage of enriched GO categories for genes regulated at the level of alternative splicing by (A) insulin and (B) wingless.
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