Comparative analysis of processed ribosomal protein pseudogenes in four mammalian genomes
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* Corresponding author: Mark Gerstein mark.gerstein@yale.edu
1 Department of Molecular Biophysics and Biochemistry, Yale University, 266 Whitney Avenue, New Haven, CT 06520, USA
2 The Saul R Korey Department of Neurology, Albert Einstein College of Medicine, NY 10461, USA
3 Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, Cambridgeshire, CB10 1HH, UK
4 Department of Computer Science, Yale University, New Haven, CT 06520, USA
5 Program in Computational Biology and Bioinformatics, Yale University, New Haven, CT 06520, USA
Genome Biology 2009, 10:R2 doi:10.1186/gb-2009-10-1-r2
Published: 5 January 2009Additional files
Additional data file 1:
Figures 5 and 6: the variation in the number of pseudogenes identified when the percent identity cutoff and e-value cutoff is varied. Figure 7: the results of manual annotation of the RPS27L/Rps27l locus in human and mouse.
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Additional data file 2:
Processed pseudogenes associated with each RP gene for human, mouse, chimpanzee and rat.
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