Research
Paternally biased X inactivation in mouse neonatal brain
1 Deptartment of Molecular Biology and Genetics, Cornell University, 227 Biotechnology Building, Ithaca, NY 14853, USA
2 Cornell Center for Comparative and Population Genomics, 130 Biotechnology Building, Cornell University, Ithaca, NY 14853, USA
3 Division of Nutritional Sciences, College of Agriculture and Life Sciences, 211 Weill Hall, Cornell University, Ithaca, NY 14853, USA
Genome Biology 2010, 11:R79 doi:10.1186/gb-2010-11-7-r79
Published: 27 July 2010Additional files
Additional file 1:
Figure S1. Distribution of imprinting status of 5,000 genes covered by the RNA-seq study.
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Additional file 2:
Table S1. Kolmogorov-Smirnov tests of p1-p2 distribution of different chromosome pairs.
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Additional file 3:
Table S2. Gene selection for pyrosequencing.
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Additional file 4:
Table S3. Least squares means (LS means) of fixed effect genes and mother.
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Additional file 5:
Figure S2. Allele-specific expression ratio of 20 genes in P2 brains of 11 female mice from each of the two reciprocal crosses between B6 and CAST strains.
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Additional file 6:
Table S4. Nonparametric analysis of variance table of the PWD × AKR data of X-linked genes subject to X inactivation.
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Additional file 7:
Table S5. Variance component analysis.
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Additional file 8:
Table S6. Analysis of variance table of the pooled data (PWD × AKR and B6 × CAST crosses) of X-linked genes subject to X inactivation. Type III sums of squares are reported.
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Additional file 9:
Figure S3. Estimation of the number of brain-forming cells at the time of X inactivation in mouse.
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Additional file 10:
Data S1. Raw pyrosequencing data summary table.
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