Table 1

Publication

Findings

Cellular context


Tay et al. [2]

Computational predictions combined with experimental analyses reveal protein-coding ceRNAs that act as trans-regulators of PTEN with similar tumor-suppressive properties

Human prostate and colon cancer cell lines

Karreth et al. [3]

Transposon insertion within ZEB2 creates a new ceRNA pairing that decreases PTEN expression levels via crosstalk involving miR-181, miR-200b, miR-25 and miR-92a

Human melanoma cell lines and a mouse model of melanoma

Cesana et al. [4]

Linc-MD1, a muscle-specific long noncoding RNA, regulates muscle differentiation by acting as a ceRNA that competes with MAML1 and MEF2C, transcripts encoding essential transcription factors involved in myogenic differentiation, for miR-133 and miR-135

Various mouse and human muscle cell types

Sumazin et al. [5]

Systematic analysis of genome-wide microRNA expression profiles reveals a ceRNA network that regulates key drivers of gliomagenesis through distinct oncogenic pathways and that determines tumor subtype formation

Human glioblastoma cell lines


Marques et al. Genome Biology 2011 12:132   doi:10.1186/gb-2011-12-11-132