gb-2011-12-11-4121465-6906 Correction <p>Correction: Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases</p> FukudaEriefukuda@ims.u-tokyo.ac.jp KaminskaHKatarzynakkaminska@genesilico.pl BujnickiMJanusziamb@genesilico.pl KobayashiIchizoikobaya@ims.u-tokyo.ac.jp

Laboratory of Social Genome Sciences, Department of Medical Genome Sciences, University of Tokyo, 4-6-1 Shirokanedai, Minato-ku, Tokyo, 108-8639, Japan

Graduate Program in Biophysics and Biochemistry, Graduate School of Science, University of Tokyo, 4-6-1 Shirokanedai, Minato-ku, Tokyo, 108-8639, Japan

International Institute of Molecular and Cell Biology, Trodena 4, 02-109 Warsaw, Poland

Faculty of Biology, Adam Mickiewicz University, Umultowska 89, 61-614 Poznan, Poland

Institute of Medical Science, University of Tokyo, 4-6-1 Shirokanedai, Minato-ku, Tokyo, 108-8639, Japan

 Genome Biology 1465-6906 2011 12 11 412 http://genomebiology.com/2011/12/11/412 10.1186/gb-2011-12-11-412 16112011231120111512012 2012BioMed Central Ltd.

Correction

We have noticed an error in the sixth paragraph of the Discussion of our article 1 . The corrected paragraph should read:

Another type IV nuclease, GmrSD, found in an E. coli strain targets glucosylated hydroxymethyl-C and may have evolved to cut T4 genome [78]. The resistance of hydroxymethyl-C-containing phage to restriction enzymes but its sensitivity to McrBC [79], the resistance of glucosylated hydroxymethyl-C to McrBC but sensitivity to GmrSD [78], and inhibition of GmrSD by T4-coded internal protein [78] suggest an evolutionary arms race (evolutionary struggle between competing sets of co-evolving genes that develop adaptations and counter-adaptations against each other) between the bacteria and the phage.

 <p>Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases.</p>FukudaEKaminskaKHBujnickiJMKobayashiIGenome Biology20089R16310.1186/gb-2008-9-11-r163261449519025584